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The dose-dependent activation of the FAK-paxillin pathway increases tendon fibroblast migration and survival under oxidative stress
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By using this setting, our study demonstrated a striking difference in the ability of B cells from RRMS patients to differentiate into both CD24 + CD38hi transitional-immature and CD24low/negCD38hi plasmablast-like B reg subsets, as compared to matched controls (Giacomini et al., 2017)
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