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Respiratory allergy and immune-mediated airway inflammation Allergic airway diseases such as asthma are driven by Th2-dominant responses and eosinophilic infiltration, traditionally regarded as archetypal “type II inflammation.” Emerging evidence identifies the Irg1-itaconate axis as a pivotal metabolic regulator restraining this process across macrophages, T cells, epithelial cells, and eosinophils, forming a systemic anti-inflammatory network
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