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An early consequence of infection is the induction of DNA damage and activation of the DNA damage response, which limits the efficiency of growth transformation

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However, based on our findings, it is more likely that the reported rescue of HRASV12-induced replication stress by TBP depletion results from downregulation of the NOX1/4 enzymes, which would be expected in the case of a deficiency in a general transcription factor such as TBP