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Set against the general role of Nrf2 as an inhibitor of carcinogenesis, it is possibly counterintuitive that somatic mutations resulting in its constitutive activation are a prominent feature of many cancers, a characteristic that is often referred to as the “dark side” of Nrf2 [382] and implies that the transcription factor is capable of supporting tumorigenesis [383, 384]

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In a 2022 review (Padeken et al., 2022), Padeken highlighted emerging evidence that H3K9me3 is upregulated by oxidative stress and proposed that the alternation of epigenetic landscape in specific tissues may result from a long-term adaptation to stress