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While a substantial body of experimental work has clarified many aspects of glial activation, intercellular communication, and their involvement in neuropathological conditions, the strength and generalizability of this evidence vary considerably across model systems and pathological contexts
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GHK-Cu has been studied for its interactions with cellular membranes, extracellular matrix remodeling, and mitochondrial function, often explored in contexts involving wound healing, neurodegenerative research, and cellular aging models
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